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  • 1
    Electronic Resource
    Electronic Resource
    Myo-, neuro-, gastrointestinal encephalopathy (MNGIE syndrome) due to partial deficiency of cytochrome-c-oxidase (1987)
    Springer
    Acta neuropathologica 74 (1987), S. 248-258 
    Details
    ISSN: 1432-0533
    Keywords: Myo-, neuro-, gastrointestinal encephalopathy (MNGIE) syndrome ; Cytochrome-c-oxidase
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary A 42-year-old woman had a 10-year history of external ophthalmoplegia, malabsorption resulting in chronic malnutrition, muscle atrophy and polyneuropathy. Computer tomography revealed hypodensity of her cerebral white matter. A metabolic disturbance consisted of lactic acidosis after moderate glucose loads with increased excretion of hydroxybutyric and fumaric acids. Post-mortem studies revealed gastrointestinal scleroderma as the morphological manifestation of her malabsorption syndrome, ocular and skeletal myopathy with ragged red fibers, peripheral neuropathy, vascular abnormalities of meningeal and peripheral nerve vessels. Biochemical examination of the liver and muscle tissues revealed a partial defect of cytochrome-c-oxidase (complex IV of the respiratory chain). This mitochondrial multisystem disorder may represent a separate entity to be classified between the spectrum of myoencephalopathies and oculo-gastrointestinal muscular dystrophy.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00688189
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  • 2
    Electronic Resource
    Electronic Resource
    Hodgkin's disease and a mediastinal tumor (1983)
    Springer
    European journal of pediatrics 141 (1983), S. 117-119 
    Details
    ISSN: 1432-1076
    Keywords: Hodgkin's disease ; Mediastinal mass ; Thymic cyst
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Two children are presented with Hodgkin's disease and a mediastinal mass which did not respond to polychemotherapy and radiation therapy and proved to be a benign thymic cyst. The coincidence of Hodgkin's disease with a benign thymic cyst has only been reported once before [11]. The diagnostic and therapeutic implications are discussed.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00496803
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  • 3
    Electronic Resource
    Electronic Resource
    Der linke Ventrikel bei chronischem Cor pulmonale (1975)
    Springer
    Virchows Archiv 366 (1975), S. 237-247 
    Details
    ISSN: 1432-2307
    Keywords: Chronic Cor pulmonale ; Left Ventricle ; Hypertrophy ; Dilatation
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Zusammenfassung In 25 Fällen von chronischem Cor pulmonale wurden die Inhalte beider Ventrikel sowie die Gewichte des freien Anteils des rechten Ventrikels und die Gewichte des linken Ventrikels mit gesamtem Septum bestimmt. Das Gewicht des freien Anteils des rechten Ventrikels lag in allen Fällen über 100 g. Der rechte Anteil des Ventrikelseptums zeigte immer eine deutliche Hypertrophie. Die Gewichte des linken Ventrikels mit dem gesamten Septum sind deshalb bei chronischem Cor pulmonale oft erhöht. Mit einer von Müller angegebenen Methode wurde das relative Gewicht des rechtsseitigen Anteils des Kammerseptums berechnet. Dieser Wert wurde von dem Gewicht des linken Ventrikels mit gesamtem Septum abgezogen. Nach dieser Korrektur zeigte sich nur dann eine Gewichtsvermehrung des linken Ventrikels, wenn eine arterielle Hypertonie oder eine Arterio- und Arteriolosklerose der Nieren festgestellt wurde. In einem Fall mit Linksherzhypertrophie konnte ein Hypertonus nicht ausgeschlossen werden. In der Mehrzahl der Fälle wurde eine deutliche Dilatation des linken Ventrikels nachgewiesen. Die stärksten Dilatationsgrade fanden sich besonders bei Patienten mit Bronchiektasen der Lungen. Unsere Befunde können dafür sprechen, daß bei chronischem Cor pulmonale ein vergrößertes Shuntvolumen über bronchopulmonale Anastomosen für die Entstehung der strukturellen Dilatation des linken Ventrikels eine Rolle spielt.
    Notes: Summary The volume of both ventricles was estimated in 25 hearts with chronic cor pulmonale. Also, the weight of the left ventricle, including the whole septum and the free part of the right ventricle, was determined to be 100 g. Distinct hypertrophy of the right ventricular part of the septum could be demonstrated in all cases. A weight increase of the left ventricle, including the whole septum, is frequently caused by the hypertrophic ventricular part of the septum. The relative weight of the right ventricular portion (calculated according to Müller's method) was subtracted from the weight of the left ventricle plus the whole septum. After this weight correction, a hypertrophy of the left ventricle could only be demonstrated in cases with hypertension or renal arterio- and arteriolosclerosis. In one case of left ventricular hypertrophy, hypertension could not be ruled out. In most of the cases a distinct dilatation of the left ventricle could be found. The highest degrees of dilatation were found in patients with bronchiectases of the lungs. Our results indicate that the structural dilatation of the left ventricle may be due to an increased shunt-volume by bronchopulmonary anastomoses.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00427412
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  • 4
    Electronic Resource
    Electronic Resource
    Intramyocardial microarteriopathy (1980)
    Springer
    Virchows Archiv 388 (1980), S. 289-311 
    Details
    ISSN: 1432-2307
    Keywords: Intramyocardial microarteriopathy ; Age ; Frequency ; Risk factors ; Functional significance
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary 1. Two basic types of arterial vessels can be distinguished in the myocardium. a) Compacta or Ventricular Wall Type. Intramyocardial microarteries with ring musculature of the media. b) Papillary Muscle Type. Arteries with longitudinal musculature arranged as bundles in the broad media. 2. The manifestation of intramyocardial microarteriopathy differs in these vessel types. PAS-positive and elastica-rich cushion-like thickening of the intima mainly occurs in the compacta type. Arterial vessels of the trabecular portion additionally show hyperelastosis of intima and media. The degenerative lesions of the papillary muscle type are characterized by early degeneration of the smooth muscle cells and hyperfibroelastosis of intima and media. 3. Pathological vascular lesions in the compacta and papillary muscle type occur more frequently in the left ventricle, and increase with age. 4. Slight and medium lesions are in the majority. Severe vascular changes are seen in the 7th to 9th decade in about 5–10% (compacta type) and in 30–35% (papillary muscle type) respectively. 5. Hypertension seemed to raise the frequency of slight and medium lesions in the compacta type vessels. In the papillary muscle type the degenerative lesions frequently extend from the top to the bottom of the papillary muscles. 6. No significant difference between frequency and severity of vascular lesions in diabetics and in the control group could be demonstrated. 7. It appears that pathological vascular lesions occur less frequently in compacta type vessels in cases of high-grade stenosis due to extramural coronary artery sclerosis. 8. Impairment of the quality of myocardial circulation is only encountered in the presence of severe pathological vascular lesions.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF00430860
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  • 5
    Electronic Resource
    Electronic Resource
    2. Pathologische Anatomie des Mammacarcinoms (1977)
    Springer
    Langenbeck's archives of surgery 345 (1977), S. 39-44 
    Details
    ISSN: 1435-2451
    Keywords: Lesions, precancerous ; Carcinoma, lobular ; Papilloma ; Carcinoma, mammary ; Staging ; Präcancerose ; Carcinoma lobulare in situ ; Papillom ; Mammacarcinom ; Staging
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: ZusammenfaBung Die Morphologie und das Entartungsrisiko präcanceröser Veränderungen der Brustdrüse (intraduktale atypische Epithelproliferation, papillomatöse Proliferationen, Carcinoma lobulare in situ) werden besprochen. Das jeweils geeignetste therapeutische Vorgehen (kurzfristige Kontrolle, s. c. Mastektomie, einfache Mastektomie) wird mitgeteilt. Ein kombiniertes intraoperatives histologisches und cytologisches “staging” durch Untersuchung der axillären Lymphknoten wurde entwickelt. Ergibt sich dabei ein Stadium I, so wird lediglich die einfache Mastektomie durchgeführt. Im Stadium II wird nach Rotter-Halsted mit präziser Ausräumung auch der Apex und der interpektoralen Lymphknoten operiert.
    Notes: Summary The morphology and the risk of precancerous lesions (intraductal atypical epithelial proliferation, papillary proliferation, and carcinoma lobulare in situ) of mammary carcinoma are demonstrated and discuBed. According to the type of lesion, the surgery of choice based on short-term observation is subcutaneous mastectomy or simple mastectomy. Combined intraoperative histologic and cytologic staging, based on the investigation of the axillary lymph nodes has been developed. We prefer simple mastectomy for stage I and ablatio mammae (method of Rotter-Halsted) for stage II to extirpate as precisely as poBible the lymph nodes of the apex and the interpectoral nodes.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01305441
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  • 6
    Electronic Resource
    Electronic Resource
    When should treatment of acute experimental pancreatitis be started? (1988)
    Springer
    Research in experimental medicine 188 (1988), S. 123-129 
    Details
    ISSN: 1433-8580
    Keywords: Acute experimental pancreatitis ; Sodium taurocholate ; Pancreatic enzymes ; Pancreatic morphology ; Rat
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary Sodium taurocholate pancreatitis in the rat is a frequently used experimental model for evaluating therapeutical regimes in this disease. It is, however, uncertain when treatment should be started, as the early phase of this experimental model and thus the time when the pancreatitis really develops is unknown. Serum and pancreatic enzymes, as well as pancreatic morphology, were therefore studied 5, 30, and 60min after induction of sodium taurocholate pancreatitis. It was found that increase in serum enzymes and decrease in pancreatic enzymes and morphological changes characteristic for acute pancreatitis develop as early as 5 and 30 min after induction of pancreatitis. Thus, therapy in this model may be started shortly after induction of acute pancreatitis.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01852268
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  • 7
    Electronic Resource
    Electronic Resource
    Pulmonary complications in fatal acute hemorrhagic pancreatitis (1983)
    Springer
    Digestive diseases and sciences 28 (1983), S. 111-116 
    Details
    ISSN: 1573-2568
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Abstract Morphological changes of the lung occur frequently in fatal acute hemorrhagic pancreatitis. The pulmonary alterations are independent of mechanical ventilation and therefore not due to iatrogenic damage caused by high inspired oxygen concentrations. The histological findings are similar to those seen in the so-called shock lung syndrome. The pulmonary lesion develops progressively and three stages can be separated: early, late, and final phase. The pulmonary complications in acute hemorrhagic pancreatitis may be explained by the release of mediators such as pancreatic enzymes or free fatty acids into the blood stream. In acute hemorrhagic pancreatitis a close monitoring for shock parameters is necessary. A fall in arterial Po2 is an early indication for mechanical ventilation, including positive end-expiratory pressure.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01315139
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  • 8
    Electronic Resource
    Electronic Resource
    Ranitidine and acute experimental pancreatitis (1986)
    Springer
    Digestive diseases and sciences 31 (1986), S. 780-781 
    Details
    ISSN: 1573-2568
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01296460
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