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  • 1
    Electronic Resource
    Electronic Resource
    Chronic hypertension changes myosin isoenzyme pattern and decreases myosin phosphorylation in the rat heart
    Amsterdam : Elsevier
    Journal of Molecular and Cellular Cardiology 20 (1988), S. 875-886 
    Details
    ISSN: 0022-2828
    Keywords: Hypertension ; Myosin P-light chain phosphorylation ; Myosin isoenzymes
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Medicine
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1016/S0022-2828(88)80142-1
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    Paper (German National Licenses)
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  • 2
    Electronic Resource
    Electronic Resource
    Increased expression of angiotensin peptides in the brain of transgenic hypertensive rats
    Amsterdam : Elsevier
    Peptides 15 (1994), S. 919-926 
    Details
    ISSN: 0196-9781
    Keywords: Angiotensin I ; Angiotensin II ; Angiotensin(1-7) ; Blood pressure ; Hypertension ; Renin gene ; Tissue renin-angiotensin system
    Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics: Chemistry and Pharmacology
    Type of Medium: Electronic Resource
    URL: http://linkinghub.elsevier.com/retrieve/pii/0196-9781(94)90051-5
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  • 3
    Electronic Resource
    Electronic Resource
    Electrolyte intake and blood pressure: A study in contradictions and controversy (1985)
    Springer
    Journal of molecular medicine 63 (1985), S. 788-792 
    Details
    ISSN: 1432-1440
    Keywords: Sodium ; Calcium ; National Health and Nutrition Examination Survey (NHANES) ; Hypertension
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Notes: Summary The widely accepted recommendation that hypertensive subjects benefit from a reduction of sodium intake has lately been challenged by a number of publications. From one analysis of the First National Health and Nutrition Examination Survey (NHANES) in the USA, the conclusion was reached that hypertension was associated more frequently with low nutritional sodium intake and low calcium intake. Other authors analysing the same data but using different criteria and statistical methods did not confirm such conclusions. The criticisms of epidemiological data concerning the relationship between salt intake and hypertension include frequently inconsistent definition of hypertension, failure to consider methodological uncertainties in the measurement of salt intake and excretion and inadequate control of confounding variables such as age, race, sex, body mass index and lifestyle. The claimed link between nutritional calcium and blood pressure is completely unclear and needs careful investigation. A reduction of sodium intake from the present day excessive amounts to moderate intakes of 3–6 g per day is still recommended in order to prevent the establishment of high blood pressure, to reduce hypertensive blood pressure levels or to reduce the doses of antihypertensive drugs. With mild hypertension being the main problem of high blood pressure management, further research is necessary to place dietary intervention in the non-pharmacological treatment of hypertension on a firmer, more rational footing.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01732282
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  • 4
    Electronic Resource
    Electronic Resource
    Increased blood pressure responses to central angiotensin II in spontaneously hypertensive rats (1978)
    Springer
    Journal of molecular medicine 56 (1978), S. 47-49 
    Details
    ISSN: 1432-1440
    Keywords: Blutdruck ; Angiotensin II ; Propranolol ; Spontan hypertensive Ratten ; Gehirn ; Blood pressure ; Angiotensin II ; Propranolol ; Spontaneously hypertensive rats ; Brain
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Summary The blood pressure responses following infusions of angiotensin II (ANG II) into the brain ventricles (i.v.t.) have been tested in spontaneously hypertensive (SH) rats and in normotensive Wistar Kyoto (WK) rats. The mean arterial blood pressure increases were significantly higher in SH rats than in WK rats. Propranolol treatment reduced blood pressure increases to i.v.t. ANG II in WK, but not in SH rats. The higher sensitivity to i.v.t. ANG II in SH rats supports a role of central ANG II in the maintenance of high blood pressure in SH rats.
    Notes: Zusammenfassung Angiotensin II (ANG II) wurde spontan hypertensiven (SH) Ratten und normotensiven Wistar Kyoto (WK) Ratten in den lateralen Hirnventrikel (i.v.t.) infundiert. Der mittlere arterielle Blutdruck stieg bei den SH Ratten signifikant höher an als bei den normotensiven WK Ratten. Propranolol-Vorbehandlung reduzierte die Blutdruckanstiege nach i.v.t. ANG II Infusionen bei WK Ratten, nicht aber bei SH Ratten. Die höhere ANG II Empfindlichkeit der SH Raten bestätigt frühere Ergebnisse, die darauf hinweisen, daß zentrales ANG III an der Aufrechterhaltung des hohen Blutdruckes von SH Ratten beteiligt sein kann.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01477452
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  • 5
    Electronic Resource
    Electronic Resource
    Humoral and neurohormonal aspects of blood pressure regulation: Focus on angiotensin (1978)
    Springer
    Journal of molecular medicine 56 (1978), S. 31-41 
    Details
    ISSN: 1432-1440
    Keywords: Renin ; Angiotensin ; Neurohormone ; Gehirn ; Blutdruck ; Renin ; Angiotensin ; Neurohormones ; Brain ; Blood-Pressure
    Source: Springer Online Journal Archives 1860-2000
    Topics: Medicine
    Description / Table of Contents: Summary Angiotensin circulates in the blood as a hormone. Its main target organs are vascular smooth muscle, adrenal gland and the kidney. Hormonal angiotensin increases blood pressure by its vasoconstrictor action, by stimulation of aldosterone secretion and subsequent sodium and water retention, and by the stimulation of catecholamine release. Circulating plasma angiotensin also effects brain mechanisms of blood pressure regulation. In addition to this hormonal function, angiotensin is present in the brain as part of an endogenous brain renin-angiotensin system. Brain angiotensin is not secreted into the blood and can be considered a neurohormone with local function. A role of brain angiotensin in the maintenance of high blood pressure of spontaneously hypertensive rats has been demonstrated. Circulating plasma angiotensin appears to influence brain renin levels and vice versa. Stimulation of specific areas in the brain known to be involved in the regulation of the cardiovascular system, stimulate renin secretion from the kidney. The renin-angiotensin system can therefore serve as an example for the intimate interrelationship between humoral and neurohumoral mechanisms of blood pressure regulation.
    Notes: Zusammenfassung Angiotensin zirkuliert als Hormon im Blut. Seine wesentlichen Zielorgane sind die glatte Gefäßmuskulatur, die Nebenniere und die Niere. Zirkulierendes hormonales Angiotensin erhöht den arteriellen Blutdruck durch Vasokonstruktion, Stimulation der Aldosteronfreisetzung und nachfolgender Salzund Wasserretention. Plasma-Angiotensin wirkt darüberhinaus auf zentrale Mechanismen der Blutdruckregulation. Angiotensin ist ebenfalls im Gehirn vorhanden als Teil des endogenen Gehirnrenin-Angiotensin-Systems. Das Gehirn-Angiotensin wird nicht an das Blut abgegeben und kann als Neurohormon mit vorwiegend lokaler Funktion angesehen werden. Eine Beteiligung des Gehirn-Angiotensins an der Aufrechterhaltung des hohen Blutdruckes spontan hypertensiver Ratten ist nachgewiesen worden. Es bestehen Rückkoppelungen zwischen dem Plasma-Angiotensin auf der einen Seite und dem Gehirn-Angiotensin auf der anderen Seite. Stimulation von bestimmten, für die zentrale Blutdruckregulation bedeutsamen Hirnarealen beeinflußt die Reninsekretion von der Niere. Das Renin-Angiotensin-System (RAS) kann als ein Modell für die engen Zusammenhänge zwischen humoraler und neurohumoraler Blutdruckregulation angesehen werden.
    Type of Medium: Electronic Resource
    URL: http://dx.doi.org/10.1007/BF01477450
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